1
However, many patients develop clinically relevant neurotoxicity, frequently resulting in treatment discontinuation.
2
Background: Anesthesia in pregnant rodents causes neurotoxicity in fetal and offspring rodents.
3
The inflammation-induced effects range from beneficial aspects of neuroprotection to detrimental neurotoxicity.
4
Toxicity was acceptable with only 1 treatment drop-out due to reversible neurotoxicity.
5
What are the underlying biological susceptibilities that lead to dependence and neurotoxicity?
6
Drug-associated neurotoxicity occurs less often than with other commonly used vinca alkaloid compounds.
7
Eight patients had resolution of neurotoxicity and three had partial improvement.
8
Manganese neurotoxicity has been extensively studied since its discovery in highly exposed workers.
9
Cationic fluxes resulting from glutamate receptor activity have recently been implicated in neurotoxicity.
10
Phosphoproteins identified in this study can potentially serve as biomarkers for 1-BP-induced neurotoxicity.
11
Here, we provide the first study on the molecular mechanism underlying Sb-associated neurotoxicity.
12
Developmental neurotoxicity due to arsenic likely results in permanent changes in brain functions.
13
Numerous methods to prevent neurotoxicity have thus far proven unsuccessful.
14
Thus, inhibition of PKCδ may be a useful target for protection against MA-induced neurotoxicity.
15
However, there is no effective therapy to circumvent the neurotoxicity of anti-cancer drug treatment.
16
Peripheral neurotoxicity is a crucial side effect of chemotherapeutic agents.
